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The pathophysiology of nephritic syndrome is a complex one, requiring multiple disciplines to effectively treat the patient. It is essential to identify the precise glomerular disease based on clinical, biologic, and histologic findings. In children, a recent skin or throat infection may suggest a glomerulonephritic infection celebmix. Arthritis or systemic lupus erythematosus may be the cause. Measurement of serum complement levels and other lab tests may be helpful for classification and follow-up.
Acute and chronic forms of nephritic syndrome are associated with various symptoms. The primary symptoms of nephrotic syndrome are glomerular hematuria and proteinuria in the subnephrotic range. Other signs include a rise in serum creatinine and systemic hypertension. While these symptoms are often associated with kidney failure, they can also be a symptom of other medical conditions.
The pathophysiology of nephritic syndrome involves the sympathetic nervous system, arginine vasopressin, and renin angiotensin aldosterone systems. The increased SNS outflow in nephritic patients is a consequence of decreased intravascular volume. This process is characterized by the Starling equation, which describes the regulation of fluid movement across the capillary walls.
The kidneys filter blood with glomeruli articlesubmit. When these are damaged, excess protein is leaked into the urine. The protein is then excreted through the urine. While the kidneys should filter excess water, too much protein causes nephrotic syndrome. It can affect both children and adults. The most common systemic disease is diabetes. Diabetes affects the kidney’s glomeruli, which can cause nephritic syndrome.
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